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Integrazione L'azione fisiologica, i protocolli d'assunzione, le materie prime ed i prodotti in commercio. Discutiamone senza censure e veti commerciali
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(#46)
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All the Truth Member
Messaggi: 4,058
Data registrazione: Jan 2007
Età: 34
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29-04-2009, 08:49 PM
Ho trovato un prodotto; AST Sports Science - Lean Muscle Tissue Stimulator - NAC 500 con questi componenti: Supplement Facts Serving Size: 2 capsules Servings per Container: 50 Ingredient Amount N-Acetyl Cysteine 500mg Manufacturer Directions: As a dietary supplement, take 1 to 2 capsules immediately after training. Manufacturer Ingredients: Other Ingredients: microcrystalline celluose, vegetable grain, magnesium stearate. ___________________________ Dovrebbe essere acetylcisteina pura... l'unica cosa è che la stessa casa ha altri due prodotti uguali.. non riesco a capire la differenza... |
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(#47)
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One of Us
Messaggi: 62
Data registrazione: Apr 2009
Età: 42
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29-04-2009, 10:12 PM
sembra un buon prodotto senza zuccheri aggiunti ...come si vede lo consigliano nei pressi di un allenamento il momento in cui una persona in salute dovrebbe assumerlo (parere personale) |
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(#48)
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All the Truth Member
Messaggi: 4,058
Data registrazione: Jan 2007
Età: 34
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30-04-2009, 04:01 PM
Quote:
post work prendo gia una caps di blend di antiox, se prendo prima dello spuntino che faccio 1 caps di questa e dopo lo spuntino gli antioss? |
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(#49)
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One of Us
Messaggi: 62
Data registrazione: Apr 2009
Età: 42
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30-04-2009, 06:23 PM
non ho capito bene il tutto da come ho compreso ti alleni poi fai lo spuntino...se cosi' fosse prenderei il tutto subito dopo allenamento e assieme allo spuntino ...comunque il discorso integratori è un discorso a mio parer molto delicato molto spesso per qualcuno potrebbe essere contraddittorio e perchè no deliterio e si puo' pure essere fraintesi anche solo per poter dare un consiglio bisognerebbe sempre tener conto di un infinita' di fattori che vanno dagli orari ,da come uno dorme,dal lavoro,da come uno si alimenta ,quanto prima del wo uno mangia la vita sociale ecc ecc e non da meno come uno si allena e da cosa si aspetta dall allenamento cioè ci sono un infinita' di variabili molto sogettive da una parte tutto puo' essere giusto come no ... |
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(#50)
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One of Us
Messaggi: 62
Data registrazione: Apr 2009
Età: 42
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30-04-2009, 06:36 PM
riguardo hai costi a volte basta andar a prendere in farmacia la sostanza madre e si risparmia molto ! gia dal fluimucil al ratiopharm che sono entrambe in farmacia contenenti l acetilcisteina uno risparmia 2 euro e la funzione e contenuto è lo stesso per entrabi ...non sempre pero' molte volte in farmacia ci si puo' trovar anche peggio come prezzo o magari dalle quantita' quasi ininfluenti .. aver una certa conoscenza ti permette di aver un ampio spettro su come prendere una decisione su una qualche sostanza/prodotto in maniera da spender al meglio i propri soldi e perchè no magari risparmiando!!! voglio far una breve precisazione NON sacrificherei mai la qualita' anche se cio' dovesse costarmi qualcosina di piu' |
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(#51)
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All the Truth Member
Messaggi: 4,058
Data registrazione: Jan 2007
Età: 34
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30-04-2009, 08:45 PM
Oggi in farmacia ho preso le compresse effervescenti da 600mg di acetilcisteina della ratiopharm..costo 10 euri per 30 compresse... potrebbe essere funzionale assumerla postwo solo nei giorni di work per risparmiare qualche capsula? subito dopo work nac e dopo spuntino antioss.. comunque con 10 euro in piu compro il barattolo da 100 in rete... Mi alleno, e di solit faccio spuntino carbo+prote senza prendere polveri e simili... vita sociale beh scuola, dormo sempre piu o meno 7/8 ore, cerco di stare sempre il piu rilassato...dall'allanemento mi aspetto miglioramenti in termini di massa ovviamente... finisco di pranzare piu o meno alle 14.30 e mi alleno 1 ora dopo piu o meno... non posso far altro a causa della scuola.... integro con tè verde blend di antiossidanti (vit a(carot) 25000UI, vit e(d-Alpha) 400IU, vitc(1000mg) acido folico(490mcg) selenio (200mcg) ne prendo una caps al mattino e una alla sera o quando mi alleno post wo... il tè(50% ecgc) prima/dopo di pranzare... questo giusto per farti un quadro generico. |
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(#52)
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All the Truth Member
Messaggi: 630
Data registrazione: Mar 2008
Età: 52
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01-05-2009, 08:26 AM
Scusate ma se mi sembra di aver capito bene che il nac svolge, tra le altre cose, anche una funzione Anti-Infiammatoria , non dovrebbe essere preso lontano dagli allenamenti ? Correggetemi se sbaglio |
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(#53)
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UncensoredModerator
Messaggi: 2,858
Data registrazione: Jun 2008
Località: Faenza
Età: 48
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01-05-2009, 11:32 AM
@Vento: avrà anche un leggero effetto antiinfiammatorio ma non pregiudica certo l'allenamento!!! Alla fine dei conti è maggiore il guadagno in antiox che la perdita per antiinfiammazione...di parecchio! |
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(#54)
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UncensoredModerator
Messaggi: 2,858
Data registrazione: Jun 2008
Località: Faenza
Età: 48
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01-05-2009, 11:44 AM
Gustatevi questo bell'abstract e poi tutti a comprare il nac Abstract N-acetylcysteine (NAC), an antioxidant and a precursor of glutathione, is currently in clinical use for various pathological conditions. No data is available as to the relationship between NAC and muscular cell proliferation or muscular degenerative disease. In this study, we assessed the effect of NAC on growth of L6 myoblasts, a rat skeletal muscle cell line, under normal or bupivacaine-treated condition. Of interest, under normal growth conditions, NAC treatment concentration-dependently increased viability, cell number, and DNA incorporation of L6 cells. Remarkably, NAC treatment for 12 to 24 h led to increased phosphorylation of ERKs, a family of mitogen-activated protein kinase known to involve in cell proliferation, in L6 cells, and specific inhibition of ERKs by PD98059, a selective inhibitor of ERKs, greatly abolished the ability of NAC to increase the number of L6 cells. More importantly, pretreatment with NAC effectively blocked decrease in the number and ERKs phosphorylation in L6 cells induced by the exposure of bupivacaine, a local anesthetic with myotoxicity. These results collectively suggest that NAC has muscular cell proliferative and protective effects and the effects by NAC appear to be, in part, mediated via increase in ERKs activation. A questo link c'è la bibliografia: ScienceDirect - Life Sciences : Muscular cell proliferative and protective effects of N-acetylcysteine by modulating activity of extracellular signal-regulated protein kinase |
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(#55)
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UncensoredModerator
Messaggi: 2,858
Data registrazione: Jun 2008
Località: Faenza
Età: 48
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01-05-2009, 11:54 AM
...e questo che mette in relazione glutammina e NAC...(prendetelo con le pinze perchè cmq si riferisce ad un prodotto commerciale!!!!) Journal of Applied Physiology, 87:4, 1999, 1381-1385. Excerpt with permission of the American Physiological Society ©1999 Effect of Supplementation With a Cysteine Donor on Muscular Performance Lands, L. C., V. L. Grey, and A. A. Smountas. Effect of supplementation with a cysteine donor on muscular performance. J. Appl. Physiol. 87(4): 1381-1385, 1999.- Oxidative stress contributes to muscular fatigue. GSH is the major intracellular antioxidant, the biosynthesis of which is dependent on cysteine availability. We hypothesized that supplementation with a whey-based cysteine donor [Immunocal (HMS90)] designed to augment intracellular GSH would enhance performance. Twenty healthy young adults (10 men, 10 women) were studied presupplementation and 3 mo postsupplementation with either Immunocal (20 g/day) or casein placebo. Muscular performance was assessed by whole leg isokinetic cycle testing, measuring peak power and 30-s work capacity. Lymphocyte GSH was used as a marker of tissue GSH. There were no baseline differences (age, ht, wt, % ideal wt, peak power, 30-s work capacity). Follow-up data on 18 subjects (9 Immunocal, 9 placebo) were analyzed. Both peak power [13 ± 3.5 (SE) %, P < 0.02] and 30-s work capacity (13 ± 3.7%, P < 0.03) increased significantly in the Immunocal group, with no change (2 ± 9.0 and 1 ± 9.3%) in the placebo group. Lymphocyte GSH also increased significantly in the Immunocal group (35.5 ± 11.04%, P < 0.02), with no change in the placebo group (-0.9 ± 9.6%). This is the first study to demonstrate that prolonged supplementation with a product designed to augment antioxidant defenses resulted in improved volitional performance. L. C. LANDS, V. L. GREY, AND A. A. SMOUNTAS OXIDATIVE STRESS contributes to the development of muscular fatigue (27). GSH is a major intracellular antioxidant, the biosynthesis of which depends on the intracellular availability of cysteine (1). Previous work has shown that supplementation with N-acetylcysteine can slow the onset of muscular fatigue (26). However, there are significant adverse effects with such treatment (18, 26), possibly related to elevations in extracellular cysteine (1, 24). Cysteine, in the form of glutamylcystine moieties, more readily enters into cells. Immunocal, a whey-based oral supplement with a relative abundance of glutamylcystine, has been shown to augment intracellular GSH concen-trations in vitro (5). We hypothesized that if this would occur in vivo, then supplementation with Immunocal would improve muscular performance. DISCUSSION We demonstrated that ingestion of Immunocal, at a dose of 20 g/day, resulted in a 35.5% increase in circulating lymphocyte GSH concentrations. At the same time, supplemented subjects were able to generate more power to perform more work during a 30-s maximal effort. Glutamyl amino acids can be transported into cells. In the case of glutamylcystine, this can effectively increase cellular GSH concentrations (2). Immunocal is a bovine whey protein concentrate produced by a proprietary lenient technique involving microfiltration and low-temperature pasteurization of milk. Whey protein consists of several compounds, including albumin, lactoferrin, and a-lactalbumin, which are rich in cystine (the oxidized form of cysteine) residues. Albumin and lactoferrin are also rich in glutamylcystine, which is easily transported into cells, making it a more readily available substrate for GSH biosynthesis (4, 5). Immunocal contains 2.5% cystine, compared with 0.3% for casein. We utilized lymphocyte GSH concentrations as a marker of tissue GSH levels, as evidence from animal studies has suggested that this could track tissue levels, in response to both L-2-oxothiazolidine-4-carboxylic acid, a cysteine precursor, and buthionine sulfoximine, an inhibitor of the first, rate-limiting step in GSH synthesis, -glutamylcysteine synthase. Although a small trial of L-2-oxothiazolidine-4-carboxylic acid raised lymphocyte GSH levels, it also resulted in elevations in plasma cysteine and adverse effects (24). Although some patients did complain of bloating and occasional queasiness while on Immunocal, no other complaints were noted. This study, then, is the first demonstration of a well-tolerated oral supplement that could effectively raise tissue GSH concentrations. Of more import are the functional findings. Subjects on Immunocal were able to generate greater power and increased the amount of work they could achieve during an all-out 30-s sprint. We have previously demonstrated that leg muscle 30-s isokinetic work output is a significant factor contributing to progressive exercise performance in patients with cystic fibrosis (20) and patients after lung transplant (22), independent of the effect of pulmonary impairment. Similar results have also been demonstrated in healthy adults (23). The ability to perform exercise has a significant impact on quality of life (10). Our results suggest then that the improvements in volitional performance that we measured in the laboratory can have a direct impact on functional ability. In this regard, it is intriguing that the subjects on Immunocal increased the percentage of time they spent being active. Oxidative stress is associated with strenuous muscular contraction, leading to fatigue (8, 13, 15, 16, 28, 31). However, as pointed out by both Reid and colleagues (26) and Sen (27), although biochemical parameters of oxidative stress can be altered by supplementation, it has been difficult to demonstrate improvement in muscular performance. Animal models of muscular fatigue have demonstrated a beneficial effect of pretreatment with N-acetylcysteine (29, 32). Reid and co-workers were the first to demonstrate that pretreatment with intravenous N-acetylcysteine could increase force output of the tibilais anterior in humans when electrically stimulated to fatigue at low frequencies. A recent study reported that the time to voluntary task failure (inability to maintain 80% of maximal transdiaphragmatic pressure while breathing against a resistive load) in healthy humans could be increased by the use of intravenous N-acetylcysteine (34). N-acetylcysteine can serve to maintain adequate stores of GSH through several mechanisms, including supplying cysteine for GSH biosynthesis and directly scavenging reactive oxygen species. Because N-acetylcysteine does not cross the sarcolemma or increase blood total GSH concentrations but does reduce blood GSH oxidation after exercise (28), it is likely that the results of Reid and co-workers (26) were due to N-acetyleysteine's free radical scavenging effects. The prevention of free radical-induced muscular dysfunction by free radical scavenging most likely explains the results of recent animal studies of diaphragm fatigue (32). However, other potential effects, such as improved blood flow or increased central nervous system respiratory drive, could also contribute (12). Unfortunately, N-acetlycysteine is associated with a number of adverse effects that detract from its utility as an ergogenic aid. These include blurred vision, dysphoria, and gastrointestinal discomfort. In the study by Travaline and co-workers (34), four subjects were premedicated with diphenhydramine and ranitidine to prevent the development of adverse effects. The exact mechanism(s) of how Immunocal improved muscular performance is unclear. The most obvious mechanism would be an increase in intracellular glutathione levels, leading to a decrease in oxidant-induced muscular dysfunction. Our patients increased the percentage of time spent in moderate-to-vigorous activity, so that a central effect leading to increased activity and improved neural regulation of muscular function cannot be excluded. Many of the subjects reported a sense of feeling more energetic. This feeling could relate to central mechansims but could also relate to a decrease in muscular damage from antioxidant protection, as muscle soreness and sarcolemma permeability have been linked to oxidative stress (30). Our activity questionnaire provides us with time spent in activity but does not describe how that time was spent. However, this enhanced activity could have led to a training effect. Subjects on Immunocal had a decrease in their percentage of body fat while maintaining their weight. Although this result sounds almost too good to be true, in healthy subjects plasma concentrations of cysteine and glutamine have been prognostic of subsequent changes in lean body mass (17). In patients with wasting disorders, such as cancer and human immunodeficiency virus infections, these values are reduced early on, preceding overt cachexia (7, 11). The biochemical changes seen in wasting disorders have led to the concept of a low cyst(e)ine-glutamine syndrome. In this model, hepatic catabolism of cyst(e)ine to sulfate leads to the generation of hydrogen ions, which remove bicarbonate through buffering. Bicarbonate is required for the first rate-limiting step in the conversion of ammonium to urea. Removal of bicarbonate promotes ammonia's conversion to glutamine, thus conserving nitrogen in the amino acid pool. Our results are consistent with this model of cysteine metabolism. The change in redox state resulting from augmentation of glutathione stores could also alter gene expression to promote muscle growth (6). This suggests that supplementation with a cysteine donor may favorably influence body composition toward increased muscle mass. We do not believe that the changes we saw in body composition and muscle function were simply due to augmented protein intake, as the casein-supplemented group did not demonstrate these changes. In conclusion, supplementation of healthy young adults with a whey-based oral supplement augmented lymphocyte GSH concentrations, while increasing muscular perfor-mance in these subjects. Aside from its potential as an ergogenic aid, such supplementation may have particular benefit in patients with persistent inflammatory conditions. REFERENCES 1. Anderson, M. E. Glutathione and glutathione delivery compounds. Adv. Pharmacol. 38: 65-78, 1997. 2. Anderson, M. E., and AL Meister. Transport and direct utilization of gamma-glutamylcyst(e)ine for gIutathione synthesis. Proc. Natl. Acad Sci. USA 80: 707-711, 1983. 3. Boucher, G. P., L. C. Lands, J. A. Hay, and L. Hornby. Activity levels and the relationship to lung function and nutritional status in children with cystic fibrosis. Am. J. Phys. Med. Rehab.76:311-315,1997. 4. Bounous, G., G. Batist, and P. Gold. Immunoenhancing property of dietary whey protein in mice: role of glutathione. Clin. Invest. Med. 12: 154-161, 1989. 5. Bounous, G., and P. Gold. The biological activity of undenatured dietary whey proteins: role of glutathione. Clin. Invest. Med. 14:296-309,1991. 6. Burdon, R. H. Superoxide and hydrogen peroxide in relation to mammalian cell proliferation. Free Radic. Biol. Med. 18: 775794,1995. 7. Droge, W., and E. Holm. Role of cysteine and glutathione in HIV infection and other diseases associated with muscle wasting and immunological dysfunction. FASEB J. 11: 1077-1089, 1997 8. Gohill, YL, C. Viguie, W. C. Stanley, G. A. Brooks, and Packer. Blood glutathione oxidation during human exercise. J. Appl. Physiol. 64:115-119,1988. 9. Grey, V. L., V. G. Kramer, and L. C. Lands. The determination of oxidised and reduced glutathione in circulating lymphocytes using the Cobas Mira S (Abstract). Clin. Biochem. 31: 301, 1998. 10. Guyatt, G. H., L. B. Berman, M. Townsend, S. O. Puggley, and L. W. Chambers. A measure of quality of life for clinical trials in chronic lung disease. Thorax 42: 773-778,1987. 11. Hack, V., D. Schmid, R. Breitkreutz, C. Stahl-Henning, P. Drings, R. Kinscherf, F. Taut, E. Holm, and W. Droge. Cystine levels, cystine flux, and protein catabolism in cancer eachexia, HIV/SIV infection, and senescence. FASEB J. 11: 84-92,1997. 12. Harrison, P. M., J. A. Wendon, A. E. Gimson, G. J. Alexander, and R. Williams. Improvement by acetyleysteine of hemodynamics and oxygen transport in fulminant hepatic failure. N. Engl. J. Med. 324:1852-1857, 1991. 13. Jackson, M. J., R. H. T. Edwards, and M. C. H. Symons. Electron spin-resonance studies of intact mammalian skeletal muscle. Biochim. Biophys. Acta 847:185-190, 1985. 14. Jetté, K, S. Hendricks, D. Kroetsch, H. Nielsen, P. Soucy, and P. Verdier. Guide for Anthropometric Meaurements of Canadian Adults. Ottawa, ON: Health-Welfare Canada, 1992. 15. Ji, L. L., A. Katz, R. Fu, M. Griffiths, and M. Spencer. Blood glutathione status during exercise: effect of carbohydrate supplementation. J. Appl. Physiol. 74: 788-792, 1993. 16. Kanter, M. M., L. A. Nolte, and J. O. Holloszy. Effects of an antioxidant vitamin mixture on lipid peroxidation at rest and postexercise. J. Appl. Physiol. 74: 965-969, 1993. 17. Kinscherf, R., V. Hack, T. Fischbach, B. Friedmann, C. Weiss, L. Edler, P. Bartsch, and W. Droge. Low plasma glutamine in combination with high glutamate levels indicate risk for loss of body cell mass in healthy individuals: the effect of N-acetyl-cysteine. J. Mol. Med. 74: 393-400, 1996. 18. Koch, S. M.9 A. A. Leis, D. S. Stokic, F. A. Khawli, and M. B. Reid. Side effects of intravenous N-acetylcysteine (Abstract). Am. J. Respir Crit. Care Med. 149: A321, 1994. 19. Lands, L. C., C. Gordon, O. Bar-Or, C. J. Blimkie, R. M. Hanning, N. L. Jones, L. A. Moss, C. E. Webber, W. 1VL Wilson, and G. J. Heigenhauser. Comparison of three techniques for body composition analysis in cystic fibrosis. J. Appl. Physiol. 75:162-166,1993. 20. Lands, L. C., G. J. Heigenhauser, and N. L. Jones. Analysis of factors limiting maximal exercise performance in cystic fibrosis. Clin. Sci. 83:391-397,1992. 21. Lands, L. C., L. Hornby, G. Desrochers, T. Her, and G. J. Heigenhauser. A simple isokinetic cycle for measurement of leg muscle function. J. Appl. Physiol. 77: 2506-2510, 1994. 22. Lands, L. C., A. A. Smountas, G. Messiano, L. Brosseau, H. Shennib, 1VL Charbonneau, and R. Gauthier. Maximal exercise capacity and peripheral skeletal muscle function following lung transplantation. J. Heart Lung. Transplant. 18: 113-120, 1999. 23. Makrdies, L., G. J. Heigenhauser, N. McCartney, and N. L. Jones. Maximal short term exercise capacity in healthy subjects aged 15-70 years. Clin. Sci. Lond. 69: 197-205, 1996. 24. Porta, P., S. Aebi, K Summer, and B. H. Lauterburg. L-2-oxothiazolidine-4-carboxylic acid, a cysteine prodrug: pharmacokinetics and effects on thiols in plasma and lymphocytes in humans. J. Pharmacol. Exp. Ther. 257: 331-334, 1991. 25. Putman, C. T., N. L. Jones, L. C. Lands, T. M. Bragg, M. G. Hollidge-Horvat, and G. J. Heigenhauser. Skeletal muscle pyruvate dehydrogenase activity during maximal exercise in humans. Am. J. Physiol. 269 (Endocrinol. Metab. 32): E458-E68, 1995. 26. Reid, M. B.t D. S. Stokic, S. M. Koch, F. A. Ehawli, and A. AL Leis. N-acetylcysteine inhibits muscle fatigue in humans. J. Clin, Invest. 94: 2468-2474, 1994. 27. Sen, C. K Oxidants and antioxidants in exercise. J. Appl. Physiol. 79: 675-686,1995. 28. Sen, C. K., M. Atalay, and O. Hanninen. Exercise-induced oxidative stress: glutathione supplementation and deficiency. J. Appl. Physiol. 77: 2177-2187, 1994. 29. Shindoh, C., A. DiMarco, A. Thomas, P. Manubay, and G. Supinski. Effect of N-acetylcysteine on diaphragm fatigue. J. Appl. Physiol. 68:2107-2113,1990. 30. Sjödin, B., Y. Hellsten Westing, and F. S. Apple. Biochemical mechanisms for oxygen free radical formation during exercise. Sports Med 10: 236-254, 1990. 31. Sumida, S., YL Tanaka, H. Kitao, and F. Nakadomo. Exerciseinduced lipid peroxidation and leakage of enzymes before and after vitamin E supplementation. Int. J. Biochem. 21: 835-838, 1989. 32. Supinski, G. S., D. Stofan, R. Ciufo, and AL DiMarco. N-acetylcysteine administration alters the response to inspiratory loading in oxygen-supplemented rats. J. Appl. Physiol. 82: 1119-1125, 1997. 33. Tietze, F. Enzymic method for quantitative determination of nanograrn amounts of total and oxidized glutathione: applications to mammalian blood and other tissues. Anal. Biochem. 27: 502-522,1969. 34. Traveline, J. M., S. Sudarshan, B. G. Roy, F. Cordova, V. Uyenson, and G. J. Criner. Effect of N-acetyleysteine on human diaphragm strength and fatigability. Am. J. Respir. Crit. Care Med. 156:1567-1571,1997. 35. Trump, M. E., G. J. Heigenhauser, C. T. Putman, and L. L. Spriet. Importance of muscle phosphocreatine during intermittent maximal cycling. J. Appl. Physiol. 80: 1574-1580, 1996. |
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(#56)
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One of Us
Messaggi: 62
Data registrazione: Apr 2009
Età: 42
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01-05-2009, 01:24 PM
pultroppo al di fuori del mio lavoro e dell attivita' fisica nel resto sono proprio una frana in inglese avevo 3 e in informatica è gia' tanto che riesco a scrivere sul forum ...x matti se sei in salute puoi benissimo divedere la cps di NAC ha meta' e prenderne meta' subito dopo l allenamento magari con vitamina c e acido alfa lipoico(sostanza molto valida)oppure assieme agli anti ox che prendi poi fai lo spuntino... l altra meta' la mattina seguente poi se stai fisicamente bene fino al prox allenamento non la prenderei piu'la NAC...non prenderei nessun tipo di integratore o antiox subito prima dell allenamento e durante visto che che gia' mangi poco prima (cerca di aver una dieta il piu' possibile pulita oltre alle prot anche verdura e frutta )...riguardo allo spuntino post lo farei con un po'di carbo semplici tipo miele ,pane bianco ho anche succhi di frutta se li tolleri e il piu' possibile proteine nobili tipo due uova o ricotta anche bresaola...cioè che siano assorbite abb velocemente ma non in grandi quantita' perchè probabilmente ci sono delle possibilita' che mangiar un ora prima dell allenamento pregiudichi lo stesso proprio per la digestione quindi sconsiglio di mangiar molto subito dopo l allenamento...non prenderei selenio e caps di te tutti i giorni ...x il the prenderei un infuso alla mattina al posto del latte o altro circa due volte alla settimana non di piu' o al max due settimane tutti i giorni e tre no !!volendoti dar un consiglio in linea di max come antiox con il the verde alla mattina prenderei anche 50 grammi di cioccolato puro al 99% quello puro al contrario di cosa pensa la gente fa meraviglie per la salute...e magari perchè no come spuntino a meta' mattina pompelmo con caffe (queste 4 cose se prese saltuarialmente tutte assieme anno un buon effetto termogeno con risparmio della massa)provare per credere...il pompelmo è una buona fonte di leucina hmb oltre a contener naringina per il dimagrimento e aver proprieta' disintossicanti per il fegato... |
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(#57)
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All the Truth Member
Messaggi: 4,058
Data registrazione: Jan 2007
Età: 34
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01-05-2009, 01:42 PM
Quote:
Non avevo pensato all'ottima idea di dividere la NAC... Quindi dopo l'allenamento Prendo meta' caps(quindi circa 300mg) di nac insieme agli antioss e lo spuntino... L'altra meta' della capsula il mattino dopo dopo/prima mangiato è indifferente? Quindi fino all'allenamento seguente dici di non utilizzarlo.. se prendessi 300mg ogni giorno? Per il tè verde in capsule quindi mi consigli di utilizzarlo a cicli? Ora è quasi 2 mesi che lo prendo tutti i giorni, 1 caps da 200mg... magari mi consigli di ciclizzare, ora finiti i 2 mesi magari per 2 settimane non lo prendo e quando lo riprendo fare 1 giorno sì e un giorno no? di solito dopo il work mangio pane di segale + pollo/tacchino o comunque fonte magra.. frutta e verdura ne mangio, la dieta cerco di tenerla molto pulita.. Proverò pompelmo... al mattino mangio di solito 1 cubetto di cioccolato all'85% provo a cercare di prendere quello ancora più puro.. |
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(#58)
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One of Us
Messaggi: 62
Data registrazione: Apr 2009
Età: 42
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01-05-2009, 02:03 PM
se puoi prendi il NAC prima di mangiar la mattina,per via di prenderlo tutti i giorni io sono sempre contrario è bene che il corpo non si abitui sempre alle stesse cose è logico se uno non si sente in forma magari con qualche stato influenzale allora lo integrerei anche tre volte al giorno come la vitamina c e qualcos altro ma solo fino alla fine del"malessere" se non ci sono patologie strane nei giorni non stressanti/impegnativi e bene lasciar il corpo arrangiarsi da se!! lo stesso vale per le cps di thè meglio se stai anche tre settimane poi puoi far un giorno si tre no oppure al bisogno! per la dieta cerca di variarla il piu' possibile e piu' spesso possibile in tutti gli orari ...molte persone la mattina mangiano sempre le stesse cose e non cè nulla di piu' sbagliato alterna piu' che puoi ...anche dopo allenamento varia sempre e cerca di far girar piu' colori possibili nella tua dieta ...(attento alle combinazioni nocive tipo latte e carne ho latte e pesce da evitar sempre)riguardo al cioccolato meno zuccheri aggiunti ci sono meglio è |
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(#59)
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All the Truth Member
Messaggi: 630
Data registrazione: Mar 2008
Età: 52
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01-05-2009, 02:15 PM
grazie per le risposte |
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(#60)
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All the Truth Member
Messaggi: 4,058
Data registrazione: Jan 2007
Età: 34
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01-05-2009, 02:21 PM
Quote:
grazie ronin,gentilissimo. |
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